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In nerves uncovered to 100 Hz when bathed in 10 mM glucose, the CAP failed suggesting that even within the presence of normoglycemic glucose further vitality substrate is required when the tissue energy demand increases. Such data are necessary as they show that glycogen has an important role underneath physiological conditions. The fact that glycogen supported CAP conduction within the absence of exogenously applied glucose most likely has no worth in ascribing a task to glycogen, since aglycaemia is an synthetic experimental procedure, and hypoglycemia is an iatrogenic consequence of insulin therapy that didn't exist prior to 1922 (Frier et al., 2014), thus can't be glycogen’s important function. However supplying lactate to axons when tissue power demand will increase implies a novel role for brain glycogen and suggests it acts as an energy buffer relatively than an energy reserve, whose position is not to single-handedly support conduction, but to act in concert with ambient ranges of normoglycemic glucose.

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